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Activation of the GH/IGF-1 axis by CJC-1295, a long-acting GHRH analog, results in serum protein profile

This molecular alteration was associated with inhibited vascular invasion in the growth plate, resulting in energy metabolism disorders, chondrocyte apoptosis, and impaired endochondral ossification, thereby manifesting the characteristic pathological features of TD

LPS-induced TNF- factor mediates pro-inflammatory and pro-fibrogenic pattern in non-alcoholic fatty liver disease

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While the N-terminal domain provides the site for GSH binding, the C-terminal domain contributes most of the amino acid residues that interact with various hydrophobic xenobiotic substrates