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Although HFD does not affect the interaction between SQSTM1 and LC3 or autophagosome-lysosome fusion, it impairs lysosomal function by significantly decreasing the expression of lysosomal H-transporting ATPase (ATP6V0D1) and the activity of cathepsin D (CTSD)

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Those prioritizing convenience over maximum effect might consider non-injectable options, but most serious protocols rely on subcutaneous delivery

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Taken together, these findings support the potential clinical benefit of antioxidant-based therapeutic approaches, including supplementation with glutathione precursors, as a means of mitigating oxidative damage and improving patient outcomes both during acute illness and in the post-infectious period