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Xu found that XJZ could regulate microbes and metabolites in gastric juice, and the regulation of some metabolites was mediated by microbes [17]

To further elucidate the pathophysiological role of mitochondrial dysfunction and inform treatment development, future studies should integrate omics-based profiling with dynamic neuroimaging to map the spatiotemporal relationship between mitochondrial imbalance and affective symptoms

Recent work has identified a number of genes required for ferroptosis, including those involved in lipid and amino acid metabolism

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Altered Nrf 2 signaling mediates hypoglycemia-induced blood-brain barrier endothelial dysfunction in vitro

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